Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis
Joint Authors
Liu, Huang-Jun
Liao, Hai-Han
Yang, Zheng
Tang, Qi-Zhu
Source
Issue
Vol. 2016, Issue 2016 (31 Dec. 2016), pp.1-12, 12 p.
Publisher
Hindawi Publishing Corporation
Publication Date
2016-05-12
Country of Publication
Egypt
No. of Pages
12
Main Subjects
Abstract EN
Peroxisome proliferator-activated receptor-γ (PPARγ) is a ligand-activated transcription factor belonging to the nuclear receptor superfamily, which plays a central role in regulating lipid and glucose metabolism.
However, accumulating evidence demonstrates that PPARγ agonists have potential to reduce inflammation, influence the balance of immune cells, suppress oxidative stress, and improve endothelial function, which are all involved in the cellular and molecular mechanisms of cardiac fibrosis.
Thus, in this review we discuss the role of PPARγ in various cardiovascular conditions associated with cardiac fibrosis, including diabetes mellitus, hypertension, myocardial infarction, heart failure, ischemia/reperfusion injury, atrial fibrillation, and several other cardiovascular disease (CVD) conditions, and summarize the developmental status of PPARγ agonists for the clinical management of CVD.
American Psychological Association (APA)
Liu, Huang-Jun& Liao, Hai-Han& Yang, Zheng& Tang, Qi-Zhu. 2016. Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis. PPAR Research،Vol. 2016, no. 2016, pp.1-12.
https://search.emarefa.net/detail/BIM-1115308
Modern Language Association (MLA)
Liu, Huang-Jun…[et al.]. Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis. PPAR Research No. 2016 (2016), pp.1-12.
https://search.emarefa.net/detail/BIM-1115308
American Medical Association (AMA)
Liu, Huang-Jun& Liao, Hai-Han& Yang, Zheng& Tang, Qi-Zhu. Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac Fibrosis. PPAR Research. 2016. Vol. 2016, no. 2016, pp.1-12.
https://search.emarefa.net/detail/BIM-1115308
Data Type
Journal Articles
Language
English
Notes
Includes bibliographical references
Record ID
BIM-1115308