PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance
Author
Source
Issue
Vol. 2016, Issue 2016 (31 Dec. 2016), pp.1-7, 7 p.
Publisher
Hindawi Publishing Corporation
Publication Date
2016-12-18
Country of Publication
Egypt
No. of Pages
7
Main Subjects
Abstract EN
PPARδ is a ligand-activated nuclear receptor that regulates the transcription of genes associated with proliferation, metabolism, inflammation, and immunity.
Within this transcription factor family, PPARδ is unique in that it initiates oncogenesis in a metabolic and tissue-specific context, especially in mammary epithelium, and can regulate autoimmunity in some tissues.
This review discusses its role in these processes and how it ultimately impacts breast cancer.
American Psychological Association (APA)
Glazer, Robert I.. 2016. PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance. PPAR Research،Vol. 2016, no. 2016, pp.1-7.
https://search.emarefa.net/detail/BIM-1115313
Modern Language Association (MLA)
Glazer, Robert I.. PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance. PPAR Research No. 2016 (2016), pp.1-7.
https://search.emarefa.net/detail/BIM-1115313
American Medical Association (AMA)
Glazer, Robert I.. PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance. PPAR Research. 2016. Vol. 2016, no. 2016, pp.1-7.
https://search.emarefa.net/detail/BIM-1115313
Data Type
Journal Articles
Language
English
Notes
Includes bibliographical references
Record ID
BIM-1115313