Mitochondrial Retrograde Signaling: Triggers, Pathways, and Outcomes
Joint Authors
da Cunha, Fernanda Marques
Torelli, Nicole Quesada
Kowaltowski, Alicia J.
Source
Oxidative Medicine and Cellular Longevity
Issue
Vol. 2015, Issue 2015 (31 Dec. 2015), pp.1-10, 10 p.
Publisher
Hindawi Publishing Corporation
Publication Date
2015-10-25
Country of Publication
Egypt
No. of Pages
10
Main Subjects
Abstract EN
Mitochondria are essential organelles for eukaryotic homeostasis.
Although these organelles possess their own DNA, the vast majority (>99%) of mitochondrial proteins are encoded in the nucleus.
This situation makes systems that allow the communication between mitochondria and the nucleus a requirement not only to coordinate mitochondrial protein synthesis during biogenesis but also to communicate eventual mitochondrial malfunctions, triggering compensatory responses in the nucleus.
Mitochondria-to-nucleus retrograde signaling has been described in various organisms, albeit with differences in effector pathways, molecules, and outcomes, as discussed in this review.
American Psychological Association (APA)
da Cunha, Fernanda Marques& Torelli, Nicole Quesada& Kowaltowski, Alicia J.. 2015. Mitochondrial Retrograde Signaling: Triggers, Pathways, and Outcomes. Oxidative Medicine and Cellular Longevity،Vol. 2015, no. 2015, pp.1-10.
https://search.emarefa.net/detail/BIM-1075668
Modern Language Association (MLA)
da Cunha, Fernanda Marques…[et al.]. Mitochondrial Retrograde Signaling: Triggers, Pathways, and Outcomes. Oxidative Medicine and Cellular Longevity No. 2015 (2015), pp.1-10.
https://search.emarefa.net/detail/BIM-1075668
American Medical Association (AMA)
da Cunha, Fernanda Marques& Torelli, Nicole Quesada& Kowaltowski, Alicia J.. Mitochondrial Retrograde Signaling: Triggers, Pathways, and Outcomes. Oxidative Medicine and Cellular Longevity. 2015. Vol. 2015, no. 2015, pp.1-10.
https://search.emarefa.net/detail/BIM-1075668
Data Type
Journal Articles
Language
English
Notes
Includes bibliographical references
Record ID
BIM-1075668