Corrigendum to “Peripheral Blood Mononuclear Cells as a Laboratory to Study Dementia in the Elderly”

Abstract EN

The article titled “Peripheral Blood Mononuclear Cells as a Laboratory to Study Dementia in the Elderly” [1] was found to contain a substantial amount of material from previously published articles including the following sources:(i)Manraj S.

Bhamra and Nicholas J.

Ashton, “Finding a pathological diagnosis for Alzheimer’s disease: Are inflammatory molecules the answer?: Proteomics and 2DE”, Electrophoresis, 2012.

10.1002/elps.201200161.

[2] (not cited)(ii)Kelly M.

Bakulski, Laura S.

Rozek, Dana C.

Dolinoy, Henry L.

Paulson and Howard Hu, “Alzheimer’s Disease and Environmental Exposure to Lead: The Epidemiologic Evidence and Potential Role of Epigenetics”, Current Alzheimer Research (2012) 9: 563.

10.2174/156720512800617991.

[3] (cited as reference [75])(iii)Cristina Gussago, “Il Recettore Adenosinico A2a Come Possibile Biomarcatore Nella Diagnosi Differenziale Delle Demenze Nell’anziano”, Università Degli Studi Di Milano.

10.13130%2Fgussago-cristina_phd2014-03-10.

[4] (not cited)(iv)B.

Arosio, A.

Bulbarelli, S.

Bastias Candia, E.

Lonati, L.

Mastronardi, P.

Romualdi, S.

Candeletti, C.

Gussago, D.

Galimberti, E.

Scarpini, B.

Dell Osso, C.

Altamura, M.

Maccarrone, L.

Bergamaschini, C.

D Addario, D.

Mari.

“Pin1 Contribution to Alzheimer s Disease: Transcriptional and Epigenetic Mechanisms in Patients with Late-Onset Alzheimer s Disease”, Neurodegenerative Diseases, 2012.10.1159/000333799.

[5] (cited as reference [66])(v)K.

Ando, P.

Dourlen, A.

V.

Sambo et al., “Tau pathology modulates Pin1 post-translational modifications and may be relevant as biomarker,” Neurobiology of Aging, vol.

34, no.

3, pp.

757–769, 2013.

10.1016/j.neurobiolaging.2012.08.004.

[6] (cited as reference [69])