Corrigendum to “Toll-Like Receptor 4–Myeloid Differentiation Primary Response Gene 88 Pathway Is Involved in the Shikonin Treatment of CIA by Regulating TregTh17 Expression”

Joint Authors

Dai, Qiaomei
Li, Ji
Yun, Yu
Wang, Jianwei

Source

Evidence-Based Complementary and Alternative Medicine

Issue

Vol. 2019, Issue 2019 (31 Dec. 2019), pp.1-1, 1 p.

Publisher

Hindawi Publishing Corporation

Publication Date

2019-03-07

Country of Publication

Egypt

No. of Pages

1

Main Subjects

Medicine

Abstract EN

In the article titled “Toll-Like Receptor 4–Myeloid Differentiation Primary Response Gene 88 Pathway Is Involved in the Shikonin Treatment of CIA by Regulating Treg/Th17 Expression” [1], reference [4], J.

A.

Carlsson et al.

[2], was incorrectly included in the reference list and should be replaced with M.

F.

Roelofs et al.

[3].

American Psychological Association (APA)

Dai, Qiaomei& Li, Ji& Yun, Yu& Wang, Jianwei. 2019. Corrigendum to “Toll-Like Receptor 4–Myeloid Differentiation Primary Response Gene 88 Pathway Is Involved in the Shikonin Treatment of CIA by Regulating TregTh17 Expression”. Evidence-Based Complementary and Alternative Medicine،Vol. 2019, no. 2019, pp.1-1.
https://search.emarefa.net/detail/BIM-1148872

Modern Language Association (MLA)

Dai, Qiaomei…[et al.]. Corrigendum to “Toll-Like Receptor 4–Myeloid Differentiation Primary Response Gene 88 Pathway Is Involved in the Shikonin Treatment of CIA by Regulating TregTh17 Expression”. Evidence-Based Complementary and Alternative Medicine No. 2019 (2019), pp.1-1.
https://search.emarefa.net/detail/BIM-1148872

American Medical Association (AMA)

Dai, Qiaomei& Li, Ji& Yun, Yu& Wang, Jianwei. Corrigendum to “Toll-Like Receptor 4–Myeloid Differentiation Primary Response Gene 88 Pathway Is Involved in the Shikonin Treatment of CIA by Regulating TregTh17 Expression”. Evidence-Based Complementary and Alternative Medicine. 2019. Vol. 2019, no. 2019, pp.1-1.
https://search.emarefa.net/detail/BIM-1148872

Data Type

Journal Articles

Language

English

Notes

Includes bibliographical references

Record ID

BIM-1148872