Nitric Oxide, Oxidative Stress, and p66Shc Interplay in Diabetic Endothelial Dysfunction
Joint Authors
Capogrossi, Maurizio C.
Magenta, Alessandra
Gaetano, Carlo
Martelli, Fabio
Greco, Simona
Source
Issue
Vol. 2014, Issue 2014 (31 Dec. 2014), pp.1-16, 16 p.
Publisher
Hindawi Publishing Corporation
Publication Date
2014-03-05
Country of Publication
Egypt
No. of Pages
16
Main Subjects
Abstract EN
Increased oxidative stress and reduced nitric oxide (NO) bioavailability play a causal role in endothelial cell dysfunction occurring in the vasculature of diabetic patients.
In this review, we summarized the molecular mechanisms underpinning diabetic endothelial and vascular dysfunction.
In particular, we focused our attention on the complex interplay existing among NO, reactive oxygen species (ROS), and one crucial regulator of intracellular ROS production, p66Shc protein.
American Psychological Association (APA)
Magenta, Alessandra& Greco, Simona& Capogrossi, Maurizio C.& Gaetano, Carlo& Martelli, Fabio. 2014. Nitric Oxide, Oxidative Stress, and p66Shc Interplay in Diabetic Endothelial Dysfunction. BioMed Research International،Vol. 2014, no. 2014, pp.1-16.
https://search.emarefa.net/detail/BIM-453388
Modern Language Association (MLA)
Magenta, Alessandra…[et al.]. Nitric Oxide, Oxidative Stress, and p66Shc Interplay in Diabetic Endothelial Dysfunction. BioMed Research International No. 2014 (2014), pp.1-16.
https://search.emarefa.net/detail/BIM-453388
American Medical Association (AMA)
Magenta, Alessandra& Greco, Simona& Capogrossi, Maurizio C.& Gaetano, Carlo& Martelli, Fabio. Nitric Oxide, Oxidative Stress, and p66Shc Interplay in Diabetic Endothelial Dysfunction. BioMed Research International. 2014. Vol. 2014, no. 2014, pp.1-16.
https://search.emarefa.net/detail/BIM-453388
Data Type
Journal Articles
Language
English
Notes
Includes bibliographical references
Record ID
BIM-453388
