The Lrp Family of Transcription Regulators in Archaea
Joint Authors
Charlier, Daniel
Peeters, Eveline
Source
Issue
Vol. 2010, Issue 2010 (31 Dec. 2010), pp.1-10, 10 p.
Publisher
Hindawi Publishing Corporation
Publication Date
2010-11-30
Country of Publication
Egypt
No. of Pages
10
Main Subjects
Abstract EN
Archaea possess a eukaryotic-type basal transcription apparatus that is regulated by bacteria-like transcription regulators.
A universal and abundant family of transcription regulators are the bacterial/archaeal Lrp-like regulators.
The Lrp family is one of the best studied regulator families in archaea, illustrated by investigations of proteins from the archaeal model organisms: Sulfolobus, Pyrococcus, Methanocaldococcus, and Halobacterium.
These regulators are extremely versatile in their DNA-binding properties, response to effector molecules, and molecular regulatory mechanisms.
Besides being involved in the regulation of the amino acid metabolism, they also regulate central metabolic processes.
It appears that these regulatory proteins are also involved in large regulatory networks, because of hierarchical regulations and the possible combinatorial use of different Lrp-like proteins.
Here, we discuss the recent developments in our understanding of this important class of regulators.
American Psychological Association (APA)
Peeters, Eveline& Charlier, Daniel. 2010. The Lrp Family of Transcription Regulators in Archaea. Archaea،Vol. 2010, no. 2010, pp.1-10.
https://search.emarefa.net/detail/BIM-495771
Modern Language Association (MLA)
Peeters, Eveline& Charlier, Daniel. The Lrp Family of Transcription Regulators in Archaea. Archaea No. 2010 (2010), pp.1-10.
https://search.emarefa.net/detail/BIM-495771
American Medical Association (AMA)
Peeters, Eveline& Charlier, Daniel. The Lrp Family of Transcription Regulators in Archaea. Archaea. 2010. Vol. 2010, no. 2010, pp.1-10.
https://search.emarefa.net/detail/BIM-495771
Data Type
Journal Articles
Language
English
Notes
Includes bibliographical references
Record ID
BIM-495771