Cardiovascular Changes in Animal Models of Metabolic Syndrome
Joint Authors
Schaan, Beatriz D’Agord
Irigoyen, Maria Cláudia
De Angelis, Kátia
Lehnen, Alexandre M.
Rodrigues, Bruno
Source
Issue
Vol. 2013, Issue 2013 (31 Dec. 2013), pp.1-11, 11 p.
Publisher
Hindawi Publishing Corporation
Publication Date
2013-03-14
Country of Publication
Egypt
No. of Pages
11
Main Subjects
Abstract EN
Metabolic syndrome has been defined as a group of risk factors that directly contribute to the development of cardiovascular disease and/or type 2 diabetes.
Insulin resistance seems to have a fundamental role in the genesis of this syndrome.
Over the past years to the present day, basic and translational research has used small animal models to explore the pathophysiology of metabolic syndrome and to develop novel therapies that might slow the progression of this prevalent condition.
In this paper we discuss the animal models used for the study of metabolic syndrome, with particular focus on cardiovascular changes, since they are the main cause of death associated with the condition in humans.
American Psychological Association (APA)
Lehnen, Alexandre M.& Rodrigues, Bruno& Irigoyen, Maria Cláudia& De Angelis, Kátia& Schaan, Beatriz D’Agord. 2013. Cardiovascular Changes in Animal Models of Metabolic Syndrome. Journal of Diabetes Research،Vol. 2013, no. 2013, pp.1-11.
https://search.emarefa.net/detail/BIM-496683
Modern Language Association (MLA)
Lehnen, Alexandre M.…[et al.]. Cardiovascular Changes in Animal Models of Metabolic Syndrome. Journal of Diabetes Research No. 2013 (2013), pp.1-11.
https://search.emarefa.net/detail/BIM-496683
American Medical Association (AMA)
Lehnen, Alexandre M.& Rodrigues, Bruno& Irigoyen, Maria Cláudia& De Angelis, Kátia& Schaan, Beatriz D’Agord. Cardiovascular Changes in Animal Models of Metabolic Syndrome. Journal of Diabetes Research. 2013. Vol. 2013, no. 2013, pp.1-11.
https://search.emarefa.net/detail/BIM-496683
Data Type
Journal Articles
Language
English
Notes
Includes bibliographical references
Record ID
BIM-496683