The Involvement of Mg2+ in Regulation of Cellular and Mitochondrial Functions

Joint Authors

Pilchova, Ivana
Klacanova, Katarina
Tatarkova, Zuzana
Kaplan, Peter
Racay, Peter

Source

Oxidative Medicine and Cellular Longevity

Issue

Vol. 2017, Issue 2017 (31 Dec. 2017), pp.1-8, 8 p.

Publisher

Hindawi Publishing Corporation

Publication Date

2017-07-05

Country of Publication

Egypt

No. of Pages

8

Main Subjects

Biology

Abstract EN

Mg2+ is an essential mineral with pleotropic impacts on cellular physiology and functions.

It acts as a cofactor of several important enzymes, as a regulator of ion channels such as voltage-dependent Ca2+ channels and K+ channels and on Ca2+-binding proteins.

In general, Mg2+ is considered as the main intracellular antagonist of Ca2+, which is an essential secondary messenger initiating or regulating a great number of cellular functions.

This review examines the effects of Mg2+ on mitochondrial functions with a particular focus on energy metabolism, mitochondrial Ca2+ handling, and apoptosis.

American Psychological Association (APA)

Pilchova, Ivana& Klacanova, Katarina& Tatarkova, Zuzana& Kaplan, Peter& Racay, Peter. 2017. The Involvement of Mg2+ in Regulation of Cellular and Mitochondrial Functions. Oxidative Medicine and Cellular Longevity،Vol. 2017, no. 2017, pp.1-8.
https://search.emarefa.net/detail/BIM-1195312

Modern Language Association (MLA)

Pilchova, Ivana…[et al.]. The Involvement of Mg2+ in Regulation of Cellular and Mitochondrial Functions. Oxidative Medicine and Cellular Longevity No. 2017 (2017), pp.1-8.
https://search.emarefa.net/detail/BIM-1195312

American Medical Association (AMA)

Pilchova, Ivana& Klacanova, Katarina& Tatarkova, Zuzana& Kaplan, Peter& Racay, Peter. The Involvement of Mg2+ in Regulation of Cellular and Mitochondrial Functions. Oxidative Medicine and Cellular Longevity. 2017. Vol. 2017, no. 2017, pp.1-8.
https://search.emarefa.net/detail/BIM-1195312

Data Type

Journal Articles

Language

English

Notes

Includes bibliographical references

Record ID

BIM-1195312